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CaMKK2 dysfunction may play an underlying role in this phenomenon as genetic deletion of Camkk2 in hippocampal neurons, myeloid cells and immortalized cell lines (HEK293 and HepG2) results in mitochondrial dysfunction that causes a similar switch in cellular energy generation from oxidative phosphorylation to glycolysis [43, 201, 202]

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Although speculative and requiring confirmation in clinical trials, the GLP-1R agonists may have beneficial effects on both sexes, dose adjustments during long treatment periods might not be needed, and the effect may persist after treatment termination

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